primary rabbit polyclonal antibodies anti-ep3 Search Results


rabbit anti ep3  (Santa Cruz Biotechnology)
93
Santa Cruz Biotechnology rabbit anti ep3
Rabbit Anti Ep3, supplied by Santa Cruz Biotechnology, used in various techniques. Bioz Stars score: 93/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/primary+rabbit+polyclonal+antibodies+anti-ep3/pm29847662-66-9-12?v=Santa+Cruz+Biotechnology
Average 93 stars, based on 1 article reviews
rabbit anti ep3 - by Bioz Stars, 2026-08
93/100 stars
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rabbit anti-her2 antibody clone ep3  (ZSGB Biotech)
90
ZSGB Biotech rabbit anti-her2 antibody clone ep3
Rabbit Anti Her2 Antibody Clone Ep3, supplied by ZSGB Biotech, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/primary+rabbit+polyclonal+antibodies+anti-ep3/us10788484-310-0-14?v=ZSGB+Biotech
Average 90 stars, based on 1 article reviews
rabbit anti-her2 antibody clone ep3 - by Bioz Stars, 2026-08
90/100 stars
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rabbit polyclonal anti ptger3  (Proteintech)
93
Proteintech rabbit polyclonal anti ptger3
Rabbit Polyclonal Anti Ptger3, supplied by Proteintech, used in various techniques. Bioz Stars score: 93/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/primary+rabbit+polyclonal+antibodies+anti-ep3/pmc06977703-218-81-88?v=Proteintech
Average 93 stars, based on 1 article reviews
rabbit polyclonal anti ptger3 - by Bioz Stars, 2026-08
93/100 stars
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rabbit anti ep3 antibody ptger3  (Bio-Techne corporation)
90
Bio-Techne corporation rabbit anti ep3 antibody ptger3
Rabbit Anti Ep3 Antibody Ptger3, supplied by Bio-Techne corporation, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/primary+rabbit+polyclonal+antibodies+anti-ep3/pm31253108-96-31-36?v=Bio-Techne+corporation
Average 90 stars, based on 1 article reviews
rabbit anti ep3 antibody ptger3 - by Bioz Stars, 2026-08
90/100 stars
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rabbit anti mouse ep3  (Santa Cruz Biotechnology)
94
Santa Cruz Biotechnology rabbit anti mouse ep3
Rabbit Anti Mouse Ep3, supplied by Santa Cruz Biotechnology, used in various techniques. Bioz Stars score: 94/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/primary+rabbit+polyclonal+antibodies+anti-ep3/pmc06953022-66-6-17?v=Santa+Cruz+Biotechnology
Average 94 stars, based on 1 article reviews
rabbit anti mouse ep3 - by Bioz Stars, 2026-08
94/100 stars
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rabbit anti ep3 antibody  (Alomone Labs)
90
Alomone Labs rabbit anti ep3 antibody
Simplified schematic illustration of pathways for Wnt/β-catenin, ERK/MAPK and PI3K/Akt and PGE 2 -metabolism. A) Canonical Wnt/β-catenin signaling. The engagement of the Wnt receptor, Frizzled, leads to the inhibition of the β-catenin destruction complex, composed of APC, axin and GSK3β. β-catenin thereby avoids ubiquitination and subsequent degradation, thus allowing it to translocate to the nucleus to activate an array of regulatory genes. B) The RAS/RAF/MEK/ERK MAPK pathway. Stimulation of the receptor tyrosine kinase (RTK) or G-protein coupled receptors (GPCRs) leads to sequential activation of RAS, RAF, MEK, and ERK causing modification of substrates promoting cell survival and proliferation. C) In the PI3K/Akt pathway, activation of the RTK or GPCRs leads to sequential modification of phosphatidyl inositol residues of the phospholipid bilayer. In this process, PI3K generates PIP3. PIP3 in association with PDK1 activates Akt. Akt then modulates the activity of downstream substrates including mTOR, thus promoting proliferation and cell survival. D) PGE 2 -metabolism. PGE 2 -synthesis begins with catalytic hydrolysis of membrane phospholipids by cytoplasmic phospholipase A2 (cPLA 2 ), thus releasing arachidonic acid (AA). By the action of the COX-1 and COX-2, AA is converted to prostaglandin H 2 (PGH 2 ). PGH 2 is then converted to PGE 2 by prostaglandin E synthase (PTGES). The main exporter of PGE 2 is thought to be multi-drug resistance related polypeptide 4 (MRP4). Removal of PGE 2 from the extracellular compartment around target cells occurs by diffusion to the blood stream and subsequent uptake and degradation in lung, liver or kidney endothelial cells or by import to colonic epithelial cells through the prostaglandin transporter (PGT) and subsequent degradation by 15-prostaglandin dehydrogenase (15-PGDH). Through autocrine and paracrine signaling, extracellular PGE 2 stimulates the prostaglandin receptors EP1–4. The EPs are GPCRs with EP1 being Gαq-coupled while EP2 and EP4 are Gαs-coupled. <t>EP3</t> is capable of coupling with different G-proteins including Gαi, Gαs and Gαq
Rabbit Anti Ep3 Antibody, supplied by Alomone Labs, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/primary+rabbit+polyclonal+antibodies+anti-ep3/pmc06599319-116-13-27?v=Alomone+Labs
Average 90 stars, based on 1 article reviews
rabbit anti ep3 antibody - by Bioz Stars, 2026-08
90/100 stars
  Buy from Supplier
anti ep3  (Santa Cruz Biotechnology)
93
Santa Cruz Biotechnology anti ep3
Simplified schematic illustration of pathways for Wnt/β-catenin, ERK/MAPK and PI3K/Akt and PGE 2 -metabolism. A) Canonical Wnt/β-catenin signaling. The engagement of the Wnt receptor, Frizzled, leads to the inhibition of the β-catenin destruction complex, composed of APC, axin and GSK3β. β-catenin thereby avoids ubiquitination and subsequent degradation, thus allowing it to translocate to the nucleus to activate an array of regulatory genes. B) The RAS/RAF/MEK/ERK MAPK pathway. Stimulation of the receptor tyrosine kinase (RTK) or G-protein coupled receptors (GPCRs) leads to sequential activation of RAS, RAF, MEK, and ERK causing modification of substrates promoting cell survival and proliferation. C) In the PI3K/Akt pathway, activation of the RTK or GPCRs leads to sequential modification of phosphatidyl inositol residues of the phospholipid bilayer. In this process, PI3K generates PIP3. PIP3 in association with PDK1 activates Akt. Akt then modulates the activity of downstream substrates including mTOR, thus promoting proliferation and cell survival. D) PGE 2 -metabolism. PGE 2 -synthesis begins with catalytic hydrolysis of membrane phospholipids by cytoplasmic phospholipase A2 (cPLA 2 ), thus releasing arachidonic acid (AA). By the action of the COX-1 and COX-2, AA is converted to prostaglandin H 2 (PGH 2 ). PGH 2 is then converted to PGE 2 by prostaglandin E synthase (PTGES). The main exporter of PGE 2 is thought to be multi-drug resistance related polypeptide 4 (MRP4). Removal of PGE 2 from the extracellular compartment around target cells occurs by diffusion to the blood stream and subsequent uptake and degradation in lung, liver or kidney endothelial cells or by import to colonic epithelial cells through the prostaglandin transporter (PGT) and subsequent degradation by 15-prostaglandin dehydrogenase (15-PGDH). Through autocrine and paracrine signaling, extracellular PGE 2 stimulates the prostaglandin receptors EP1–4. The EPs are GPCRs with EP1 being Gαq-coupled while EP2 and EP4 are Gαs-coupled. <t>EP3</t> is capable of coupling with different G-proteins including Gαi, Gαs and Gαq
Anti Ep3, supplied by Santa Cruz Biotechnology, used in various techniques. Bioz Stars score: 93/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/primary+rabbit+polyclonal+antibodies+anti-ep3/pmc08146234-59-24-26?v=Santa+Cruz+Biotechnology
Average 93 stars, based on 1 article reviews
anti ep3 - by Bioz Stars, 2026-08
93/100 stars
  Buy from Supplier
ep3 polyclonal antibody  (Bioss)
93
Bioss ep3 polyclonal antibody
Simplified schematic illustration of pathways for Wnt/β-catenin, ERK/MAPK and PI3K/Akt and PGE 2 -metabolism. A) Canonical Wnt/β-catenin signaling. The engagement of the Wnt receptor, Frizzled, leads to the inhibition of the β-catenin destruction complex, composed of APC, axin and GSK3β. β-catenin thereby avoids ubiquitination and subsequent degradation, thus allowing it to translocate to the nucleus to activate an array of regulatory genes. B) The RAS/RAF/MEK/ERK MAPK pathway. Stimulation of the receptor tyrosine kinase (RTK) or G-protein coupled receptors (GPCRs) leads to sequential activation of RAS, RAF, MEK, and ERK causing modification of substrates promoting cell survival and proliferation. C) In the PI3K/Akt pathway, activation of the RTK or GPCRs leads to sequential modification of phosphatidyl inositol residues of the phospholipid bilayer. In this process, PI3K generates PIP3. PIP3 in association with PDK1 activates Akt. Akt then modulates the activity of downstream substrates including mTOR, thus promoting proliferation and cell survival. D) PGE 2 -metabolism. PGE 2 -synthesis begins with catalytic hydrolysis of membrane phospholipids by cytoplasmic phospholipase A2 (cPLA 2 ), thus releasing arachidonic acid (AA). By the action of the COX-1 and COX-2, AA is converted to prostaglandin H 2 (PGH 2 ). PGH 2 is then converted to PGE 2 by prostaglandin E synthase (PTGES). The main exporter of PGE 2 is thought to be multi-drug resistance related polypeptide 4 (MRP4). Removal of PGE 2 from the extracellular compartment around target cells occurs by diffusion to the blood stream and subsequent uptake and degradation in lung, liver or kidney endothelial cells or by import to colonic epithelial cells through the prostaglandin transporter (PGT) and subsequent degradation by 15-prostaglandin dehydrogenase (15-PGDH). Through autocrine and paracrine signaling, extracellular PGE 2 stimulates the prostaglandin receptors EP1–4. The EPs are GPCRs with EP1 being Gαq-coupled while EP2 and EP4 are Gαs-coupled. <t>EP3</t> is capable of coupling with different G-proteins including Gαi, Gαs and Gαq
Ep3 Polyclonal Antibody, supplied by Bioss, used in various techniques. Bioz Stars score: 93/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/primary+rabbit+polyclonal+antibodies+anti-ep3/custom%40bs-1876r%4028844837?v=Bioss
Average 93 stars, based on 1 article reviews
ep3 polyclonal antibody - by Bioz Stars, 2026-08
93/100 stars
  Buy from Supplier
monoclonal antibodies mouse anti-ki67(mib1)  (ZSGB Biotech)
90
ZSGB Biotech monoclonal antibodies mouse anti-ki67(mib1)
Simplified schematic illustration of pathways for Wnt/β-catenin, ERK/MAPK and PI3K/Akt and PGE 2 -metabolism. A) Canonical Wnt/β-catenin signaling. The engagement of the Wnt receptor, Frizzled, leads to the inhibition of the β-catenin destruction complex, composed of APC, axin and GSK3β. β-catenin thereby avoids ubiquitination and subsequent degradation, thus allowing it to translocate to the nucleus to activate an array of regulatory genes. B) The RAS/RAF/MEK/ERK MAPK pathway. Stimulation of the receptor tyrosine kinase (RTK) or G-protein coupled receptors (GPCRs) leads to sequential activation of RAS, RAF, MEK, and ERK causing modification of substrates promoting cell survival and proliferation. C) In the PI3K/Akt pathway, activation of the RTK or GPCRs leads to sequential modification of phosphatidyl inositol residues of the phospholipid bilayer. In this process, PI3K generates PIP3. PIP3 in association with PDK1 activates Akt. Akt then modulates the activity of downstream substrates including mTOR, thus promoting proliferation and cell survival. D) PGE 2 -metabolism. PGE 2 -synthesis begins with catalytic hydrolysis of membrane phospholipids by cytoplasmic phospholipase A2 (cPLA 2 ), thus releasing arachidonic acid (AA). By the action of the COX-1 and COX-2, AA is converted to prostaglandin H 2 (PGH 2 ). PGH 2 is then converted to PGE 2 by prostaglandin E synthase (PTGES). The main exporter of PGE 2 is thought to be multi-drug resistance related polypeptide 4 (MRP4). Removal of PGE 2 from the extracellular compartment around target cells occurs by diffusion to the blood stream and subsequent uptake and degradation in lung, liver or kidney endothelial cells or by import to colonic epithelial cells through the prostaglandin transporter (PGT) and subsequent degradation by 15-prostaglandin dehydrogenase (15-PGDH). Through autocrine and paracrine signaling, extracellular PGE 2 stimulates the prostaglandin receptors EP1–4. The EPs are GPCRs with EP1 being Gαq-coupled while EP2 and EP4 are Gαs-coupled. <t>EP3</t> is capable of coupling with different G-proteins including Gαi, Gαs and Gαq
Monoclonal Antibodies Mouse Anti Ki67(mib1), supplied by ZSGB Biotech, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/primary+rabbit+polyclonal+antibodies+anti-ep3/pm30974091-51-3-10?v=ZSGB+Biotech
Average 90 stars, based on 1 article reviews
monoclonal antibodies mouse anti-ki67(mib1) - by Bioz Stars, 2026-08
90/100 stars
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xpnpep3 rabbit polyclonal antibody  (OriGene)
N/A
XPNPEP3 Antibody middle region
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ep300 rabbit polyclonal antibody  (OriGene)
N/A
Rabbit polyclonal anti p300 antibody
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dpep3 antibody - c-terminal region (oaab00805)  (Aviva Systems)
N/A
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Image Search Results


Simplified schematic illustration of pathways for Wnt/β-catenin, ERK/MAPK and PI3K/Akt and PGE 2 -metabolism. A) Canonical Wnt/β-catenin signaling. The engagement of the Wnt receptor, Frizzled, leads to the inhibition of the β-catenin destruction complex, composed of APC, axin and GSK3β. β-catenin thereby avoids ubiquitination and subsequent degradation, thus allowing it to translocate to the nucleus to activate an array of regulatory genes. B) The RAS/RAF/MEK/ERK MAPK pathway. Stimulation of the receptor tyrosine kinase (RTK) or G-protein coupled receptors (GPCRs) leads to sequential activation of RAS, RAF, MEK, and ERK causing modification of substrates promoting cell survival and proliferation. C) In the PI3K/Akt pathway, activation of the RTK or GPCRs leads to sequential modification of phosphatidyl inositol residues of the phospholipid bilayer. In this process, PI3K generates PIP3. PIP3 in association with PDK1 activates Akt. Akt then modulates the activity of downstream substrates including mTOR, thus promoting proliferation and cell survival. D) PGE 2 -metabolism. PGE 2 -synthesis begins with catalytic hydrolysis of membrane phospholipids by cytoplasmic phospholipase A2 (cPLA 2 ), thus releasing arachidonic acid (AA). By the action of the COX-1 and COX-2, AA is converted to prostaglandin H 2 (PGH 2 ). PGH 2 is then converted to PGE 2 by prostaglandin E synthase (PTGES). The main exporter of PGE 2 is thought to be multi-drug resistance related polypeptide 4 (MRP4). Removal of PGE 2 from the extracellular compartment around target cells occurs by diffusion to the blood stream and subsequent uptake and degradation in lung, liver or kidney endothelial cells or by import to colonic epithelial cells through the prostaglandin transporter (PGT) and subsequent degradation by 15-prostaglandin dehydrogenase (15-PGDH). Through autocrine and paracrine signaling, extracellular PGE 2 stimulates the prostaglandin receptors EP1–4. The EPs are GPCRs with EP1 being Gαq-coupled while EP2 and EP4 are Gαs-coupled. EP3 is capable of coupling with different G-proteins including Gαi, Gαs and Gαq

Journal: BMC Cancer

Article Title: Possible predisposition for colorectal carcinogenesis due to altered gene expressions in normal appearing mucosa from patients with colorectal neoplasia

doi: 10.1186/s12885-019-5833-8

Figure Lengend Snippet: Simplified schematic illustration of pathways for Wnt/β-catenin, ERK/MAPK and PI3K/Akt and PGE 2 -metabolism. A) Canonical Wnt/β-catenin signaling. The engagement of the Wnt receptor, Frizzled, leads to the inhibition of the β-catenin destruction complex, composed of APC, axin and GSK3β. β-catenin thereby avoids ubiquitination and subsequent degradation, thus allowing it to translocate to the nucleus to activate an array of regulatory genes. B) The RAS/RAF/MEK/ERK MAPK pathway. Stimulation of the receptor tyrosine kinase (RTK) or G-protein coupled receptors (GPCRs) leads to sequential activation of RAS, RAF, MEK, and ERK causing modification of substrates promoting cell survival and proliferation. C) In the PI3K/Akt pathway, activation of the RTK or GPCRs leads to sequential modification of phosphatidyl inositol residues of the phospholipid bilayer. In this process, PI3K generates PIP3. PIP3 in association with PDK1 activates Akt. Akt then modulates the activity of downstream substrates including mTOR, thus promoting proliferation and cell survival. D) PGE 2 -metabolism. PGE 2 -synthesis begins with catalytic hydrolysis of membrane phospholipids by cytoplasmic phospholipase A2 (cPLA 2 ), thus releasing arachidonic acid (AA). By the action of the COX-1 and COX-2, AA is converted to prostaglandin H 2 (PGH 2 ). PGH 2 is then converted to PGE 2 by prostaglandin E synthase (PTGES). The main exporter of PGE 2 is thought to be multi-drug resistance related polypeptide 4 (MRP4). Removal of PGE 2 from the extracellular compartment around target cells occurs by diffusion to the blood stream and subsequent uptake and degradation in lung, liver or kidney endothelial cells or by import to colonic epithelial cells through the prostaglandin transporter (PGT) and subsequent degradation by 15-prostaglandin dehydrogenase (15-PGDH). Through autocrine and paracrine signaling, extracellular PGE 2 stimulates the prostaglandin receptors EP1–4. The EPs are GPCRs with EP1 being Gαq-coupled while EP2 and EP4 are Gαs-coupled. EP3 is capable of coupling with different G-proteins including Gαi, Gαs and Gαq

Article Snippet: The following antibodies were tested: Mouse anti-EP3 antibody (5F5), sc-57,105 (Santa Cruz Biotechnology), Rabbit anti-EP3 antibody, 101,760 (Cayman Chemicals, Ann Arbor, MI, USA), Rabbit anti-EP3 antibody, APR-065 (Alomone Labs, Jerusalem, Israel), Rabbit anti-EP3 antibody (PTGER3), NBP1–84835 (Bio-techne, Abingdon, United Kingdom).

Techniques: Inhibition, Activation Assay, Modification, Activity Assay, Diffusion-based Assay

Quantitative real-time PCR primer sequences

Journal: BMC Cancer

Article Title: Possible predisposition for colorectal carcinogenesis due to altered gene expressions in normal appearing mucosa from patients with colorectal neoplasia

doi: 10.1186/s12885-019-5833-8

Figure Lengend Snippet: Quantitative real-time PCR primer sequences

Article Snippet: The following antibodies were tested: Mouse anti-EP3 antibody (5F5), sc-57,105 (Santa Cruz Biotechnology), Rabbit anti-EP3 antibody, 101,760 (Cayman Chemicals, Ann Arbor, MI, USA), Rabbit anti-EP3 antibody, APR-065 (Alomone Labs, Jerusalem, Israel), Rabbit anti-EP3 antibody (PTGER3), NBP1–84835 (Bio-techne, Abingdon, United Kingdom).

Techniques: Real-time Polymerase Chain Reaction

Difference in mRNA expression in CRN versus controls

Journal: BMC Cancer

Article Title: Possible predisposition for colorectal carcinogenesis due to altered gene expressions in normal appearing mucosa from patients with colorectal neoplasia

doi: 10.1186/s12885-019-5833-8

Figure Lengend Snippet: Difference in mRNA expression in CRN versus controls

Article Snippet: The following antibodies were tested: Mouse anti-EP3 antibody (5F5), sc-57,105 (Santa Cruz Biotechnology), Rabbit anti-EP3 antibody, 101,760 (Cayman Chemicals, Ann Arbor, MI, USA), Rabbit anti-EP3 antibody, APR-065 (Alomone Labs, Jerusalem, Israel), Rabbit anti-EP3 antibody (PTGER3), NBP1–84835 (Bio-techne, Abingdon, United Kingdom).

Techniques: Expressing